Project Details
Description
Traumatic brain injury (TBI) is a modifiable risk factor for dementia. Neuroinflammation is a potential driver for dementia-associated neurodegenerative pathologies after TBI, but the mechanism is unknown. During neuroinflammation, microglial cells remove protein deposits through autophagy. The aim of this project is to characterise the crosstalk of autophagy (Atg) and protein expression pathways with the induction of neuroinflammatory responses in human microglia.
| Short title | Determining the Impact of Microglial Autophagy on Neuroinflammatory Responses |
|---|---|
| Status | Active |
| Effective start/end date | 1/04/24 → 31/03/25 |
Fingerprint
Explore the research topics touched on by this project. These labels are generated based on the underlying awards/grants. Together they form a unique fingerprint.